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Likewise, parenteral administration of benzyloctadecanamide exhibited an improvement in post-ischemic damage neurobehavioral performance through recovery of somatosensory function (Yang et al., 2022) and decrease of sensory deficit (Castro, 2022). Finally, an in silico study reported interaction between glucosinolates and tryptophan and histidine residues of AchE and BuChE enzymes, suggesting that they may act by blocking the active catalytic side of these enzymes (Tarabasz et al., 2022). Within the isolated compounds, it has been suggested that macamides act through the cannabinoid receptor type 1 (CB1) due to their chemical structure similar to anandamide (endogenous cannabinoid) and by observing interaction with the nuclear receptor activated by peroxisomal proliferators gamma (PPARγ) (Gugnani et al., 2018), involved in cellular metabolism and homeostasis. 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