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Ketone supplements might also improve fueling during exercise, reduce lactate production, and improve performance due to glucose sparing (Okuda et al., 1991), and have positive effects on anxiety (Kashiwaya et al., 2013), and mental performance and memory (Kashiwaya et al., 2013). Ketone supplements demonstrate promise as potential adjunct treatments for brain injury (White & Venkatesh, 2011), cancer (Poff et al., 2015; Poff et al., 2014), Angelman syndrome (Ciarlone et al., 2016), for reducing inflammation by suppressing activation of the NLRP3 inflammasome (Youm et al., 2015), and Alzheimer’s disease (Kashiwaya et al., 2013). They are considered to be a safe and effective way to increase ketone body concentrations (Hashim & Van Itallie, 2014). When fed intravenously, MCTs increase ketogenesis when compared to both structurally similar fats (Mingrone et al., 1993) and LCTs (Jiang et al., 1993; Lai & Chen, 2000). MCTs promote ketonaemia and ketogenesis (useful to reduce the risk of night-time hypoglycaemic coma) in those with carnitine palmitoyltransferase deficiency, a rare genetic condition which inhibits the ability to produce ketone bodies from long-chain fatty acids (Bonnefont et al., 1989; Bougnères et al., 1981). Dietary MCTs are also known to promote both ketonaemia and ketogenesis in animals (Bach et al., 1977; Yeh & Zee, 1976) and humans with and without health conditions (St-Onge et al., 2003; Yajnik et al., 1997). Similar to the short-chain fatty acids and unlike long-chain triglycerides (LCTs), MCTs do not require the actions of bile, nor micellar-chylomicron mediated absorption into the lymphatics and instead are diffused directly into the hepatic portal vein and preferentially converted into bio-available ketone bodies in the liver. Both 2 g and 4 g of butyric acid were demonstrated to be more ketogenic than either 5 g of leucine, or 5 or 10 g of O-MAG (St-Pierre et al., 2017). 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