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In ADPKD, cyst linings are thought to be metabolically inflexible and reliant on glucose, thus the lowering of available glucose via a KD provides a potentially promising means of combating cyst development (24). It also reduces the release of glutamate, thereby lowering extracellular levels of this excitatory neurotransmitter (17). Mitochondrial ketolysis increases the synthesis and accumulation of GABA, an inhibitory neurotransmitter in GABAergic neurons (10). Additionally, ketone bodies have been found to play an active role in the regulation of neuronal excitability by decreasing neuronal firing. Firstly, ketone bodies act as ligands for several G protein coupled receptors, thus orchestrating cellular responses and acting as signaling molecules in pathways related to lipid metabolism and cellular growth (22). As well as providing an alternative fuel source for the brain, ketone bodies are thought to regulate the nervous system. 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