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Emerging research from animal studies has demonstrated the potential for exercise to be a viable alternative for managing certain GC-induced side effects, including skeletal muscle atrophy and insulin resistance (11, 12, 13). Subgroup analyses indicated that the effect of exercise training was not significantly affected by training mode, age, body mass status, or testosterone measure. RE-induced increases in key endogenous steroid and peptide hormone responses are likely to be an integral part of the integrated response to acute exercise and exercise-induced muscle growth. The local production of IGF-1 is controlled primarily by GH and other hormones (e.g., parathyroid and thyroid hormones) (Bikle et al., 2015); suggesting GH's effect on growth may be mediated in part via increased local IGF-1 production and/or action.