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One of the main reasons for this mutation is the increase in the level of ROS and oxidative stress in cancer patients, which causes mitochondrial damage and mutations [74,75]. Numerous experimental studies have shown that in some cancers, such as cancers of the head and neck , liver, breast, colon, ovary, and prostate, mutations occur in mitochondrial DNA and impair normal mitochondrial function. Another mechanism involved in the beneficial effects of KD on cancer cells through its effect on mitochondrial metabolism. In addition, high serum insulin levels have been shown to stimulate the gene expression of vascular endothelial growth factor (VEGF), which is an essential component of vascular angiogenesis . In fact, cancer cells use glycolysis as their main pathway of energy production to have more access to glucose and speed up glucose utilization, even in the presence of adequate amounts of oxygen, resulting in the accumulation of lactate, a process called the Warburg effect . 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