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Choline serves as a dietary precursor for the gut microbial-generated trimethylamine (TMA), which is oxidized by the hepatic enzyme flavin-containing monooxygenase 3 (FMO3) to form trimethylamine-N-oxide (TMAO) [104,105]. Although clinical evidence on the efficacy of choline supplementation in protecting against the NAFLD development and progression is lacking [102,103], the current evidence suggests the potential of choline for nutritional support and management of NAFLD. Clinical evidence on choline and weight loss is scarce, with no authoritative reports or systematic reviews specifically on choline and weight management identified. Finally, the beneficial effect of GCBE on SBP and DBP was demonstrated in a meta-analysis of nine studies in people with hypertension, whereby the effect size in SBP was larger with GCBE doses ≥400 mg/day . Regarding lipid profile, a meta-analysis of 13 randomized studies showed that GCBE or CGA between 200 and 2000 mg/day for 2–24 weeks significantly reduced fasting blood TC but not TG, LDL-C, or HDL-C . However, the results of another meta-analysis of 10 studies found a significant decrease in FBG but not in insulin, and that the decrease in FBG was only significant for GCBE ≥400 mg/day . Two meta-analyses of randomized studies showed that GCBE or CGA between 200–2000 mg/day for 2–24 weeks significantly reduced FBG and insulin, and this was not influenced by dose level [78,79]. Caffeine and CGA in GCBE can stimulate fatty acid oxidation and inhibit lipogenesis, which, in turn, decreases fat accumulation in the body . Of the studies included in four recent meta-analyses on GCBE and cardiometabolic outcomes [78,79,80,81], only seven assessed adverse events, and of these, two reported some undesirable side effects during the GCBE or CGA intervention. 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